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codelink software package  (Agilent technologies)


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    Agilent technologies codelink software package
    Codelink Software Package, supplied by Agilent technologies, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/codelink+software+package/pm17618285-207-7-12
    Average 90 stars, based on 1 article reviews
    codelink software package - by Bioz Stars, 2026-10
    90/100 stars

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    Software:

    Article Title: Loss of GLIS2 causes nephronophthisis in humans and mice by increased apoptosis and fibrosis.
    Article Snippet: Nephronophthisis (NPHP), an autosomal recessive kidney disease, is the most frequent genetic cause of end-stage renal failure in the first three decades of life.. Positional cloning of the six known NPHP genes1–4 has linked its pathogenesis to primary cilia function3,5.. Here we identify mutation of GLIS2 as causing an NPHP-like phenotype in humans and mice, using positional cloning and mouse transgenics, respectively.



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    Transcriptional profiling of KIR2D− and KIR2D+ CD8+ T cells. ( A ) Differential gene expression profile <t>(CodeLink</t> Genome Array) of KIR− (column 1), KIR2DL1/S1+ (column 2), and KIR2DL2/L3/S2+ (column 3) CD8+ T cell subsets. The figure shows the top 40 most significantly modulated genes (designed with http://www.ehbio.com/ImageGP/ ). ( B ) Total number of up- and down-regulated genes in each KIR2D+ CD8+ T cell subset (analysis performed with jvenn software). ( C ) Microarray results were confirmed with real time quantitative RT-PCR (qPCR) analysis. Expression ratio (upper plot) and relative fold changes (lower plot) are shown for microarray and qPCR assays, respectively. Differential gene expression was considered significant with p < 0.05 in three independent cells preparations. Mean fold-changes in gene transcript expression levels between KIR2D− and KIR2D+ were evaluated with 2 ΔΔCt . ( D ) Shows the normalized intensity of important molecules in the cytotoxic T cell biology and interleukin or interleukin receptors.
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    Transcriptional profiling of KIR2D− and KIR2D+ CD8+ T cells. ( A ) Differential gene expression profile <t>(CodeLink</t> Genome Array) of KIR− (column 1), KIR2DL1/S1+ (column 2), and KIR2DL2/L3/S2+ (column 3) CD8+ T cell subsets. The figure shows the top 40 most significantly modulated genes (designed with http://www.ehbio.com/ImageGP/ ). ( B ) Total number of up- and down-regulated genes in each KIR2D+ CD8+ T cell subset (analysis performed with jvenn software). ( C ) Microarray results were confirmed with real time quantitative RT-PCR (qPCR) analysis. Expression ratio (upper plot) and relative fold changes (lower plot) are shown for microarray and qPCR assays, respectively. Differential gene expression was considered significant with p < 0.05 in three independent cells preparations. Mean fold-changes in gene transcript expression levels between KIR2D− and KIR2D+ were evaluated with 2 ΔΔCt . ( D ) Shows the normalized intensity of important molecules in the cytotoxic T cell biology and interleukin or interleukin receptors.
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    Transcriptional profiling of KIR2D− and KIR2D+ CD8+ T cells. ( A ) Differential gene expression profile (CodeLink Genome Array) of KIR− (column 1), KIR2DL1/S1+ (column 2), and KIR2DL2/L3/S2+ (column 3) CD8+ T cell subsets. The figure shows the top 40 most significantly modulated genes (designed with http://www.ehbio.com/ImageGP/ ). ( B ) Total number of up- and down-regulated genes in each KIR2D+ CD8+ T cell subset (analysis performed with jvenn software). ( C ) Microarray results were confirmed with real time quantitative RT-PCR (qPCR) analysis. Expression ratio (upper plot) and relative fold changes (lower plot) are shown for microarray and qPCR assays, respectively. Differential gene expression was considered significant with p < 0.05 in three independent cells preparations. Mean fold-changes in gene transcript expression levels between KIR2D− and KIR2D+ were evaluated with 2 ΔΔCt . ( D ) Shows the normalized intensity of important molecules in the cytotoxic T cell biology and interleukin or interleukin receptors.

    Journal: Cancers

    Article Title: KIR+ CD8+ T Lymphocytes in Cancer Immunosurveillance and Patient Survival: Gene Expression Profiling

    doi: 10.3390/cancers12102991

    Figure Lengend Snippet: Transcriptional profiling of KIR2D− and KIR2D+ CD8+ T cells. ( A ) Differential gene expression profile (CodeLink Genome Array) of KIR− (column 1), KIR2DL1/S1+ (column 2), and KIR2DL2/L3/S2+ (column 3) CD8+ T cell subsets. The figure shows the top 40 most significantly modulated genes (designed with http://www.ehbio.com/ImageGP/ ). ( B ) Total number of up- and down-regulated genes in each KIR2D+ CD8+ T cell subset (analysis performed with jvenn software). ( C ) Microarray results were confirmed with real time quantitative RT-PCR (qPCR) analysis. Expression ratio (upper plot) and relative fold changes (lower plot) are shown for microarray and qPCR assays, respectively. Differential gene expression was considered significant with p < 0.05 in three independent cells preparations. Mean fold-changes in gene transcript expression levels between KIR2D− and KIR2D+ were evaluated with 2 ΔΔCt . ( D ) Shows the normalized intensity of important molecules in the cytotoxic T cell biology and interleukin or interleukin receptors.

    Article Snippet: Normalized data from the CodeLink software package were analyzed and only the genes that passed the CodeLink Bioarray quality controls were selected.

    Techniques: Gene Expression, Software, Microarray, Quantitative RT-PCR, Expressing